The first published autopsy report of a patient who died following a COVID19 vaccination was released and the implications have many people scared. The patient was an 86 year-old German man who had tested negative for COVID19 and was administered the Pfizer-BioNTech lipid nanoparticle-formulated, nucleoside-modified RNA vaccine BNT162b2 in a 30 μg dose. Eighteen days later the man was hospitalized. Six days after admission he was housed with a COVID+ patient and contracted the virus in spite of the vaccination. Two days later and a total of twenty-six days after his vaccination, the man was dead from acute kidney failure due to Arteriosclerosis and kidney damage (tubular injury), and respiratory failure from acute bronchopneumonia.
The case is detailed in the “First case of postmortem study in a patient vaccinated against SARS-CoV-2.” The doctors performing the autopsy pointed out that this case needed further examination for “lethal adverse effects.” According to DailyExpose.co.uk, “the man had a medical history including systemic arterial hypertension, chronic venous insufficiency, dementia, and prostate carcinoma.”
The data suggest that the primary cause of death was renal failure and that bronchopneumonia and COVID19 were comorbid. Given that the patient had pre-existing arteriosclerosis of the kidneys (hardening of the arteries) and according to the NIH, there exists “the strong possibility of direct damage to the kidney tubules by the SARS-CoV-2 virus” and “There is a high incidence of acute kidney injury (AKI) in patients with the severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2) infection.” This could suggest that due to the already weakened state of the patient’s kidneys they were already susceptible to the type of Acute Kidney Injury that is reportedly caused by a high viral load of COVID19 and cellular damage caused by the virus’ spike proteins.
What Does The Autopsy Ultimately Tell Us?
The study’s authors concluded: “In summary, the results of our autopsy case study in a patient with mRNA vaccine confirm the view that by first dose of vaccination against SARS-CoV-2 immunogenicity can already be induced, while sterile immunity is not adequately developed.”
An 86 year-old man who while having some medical issues commonly associated with old age, who appeared to be by no means dying, died due to the high viral load of spike proteins in his bloodstream damaging his already weakened kidneys, causing them to ultimately fail exacerbated by bronchopneumonia.
The viral load was introduced by the vaccine and intensified by the patient being exposed to infection while hospitalized. The infection appeared to be in “early stages” and had not infiltrated his body completely, as the report said, “An early stage of infection might also explain why different regions such as the olfactory bulb and liver were not (yet) affected by systemic viral spread.”
Therefore, the events that culminated in this man’s death lead to a fairly inescapable conclusion: had this man whose medical status made him particularly vulnerable to possible adverse effects of the vaccine, NOT been vaccinated, had he stayed home and simply avoided infection or even suffered from naturally acquired COVID which may not have so rapidly increased the viral load in his bloodstream damaging his kidneys, he could still be alive today. This man’s death ultimately and logically started with the vaccine.